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personal data approved: 2024. I. 11.
Personal data
Virág Vas
name Virág Vas
name of institution
doctoral school
ELTE Doctoral School of Biology (Supervisor)
the share of work in the different doctoral schools. ELTE Doctoral School of Biology 100%
Contact details
E-mail address vas.viragttk.mta.hu
phone number +36 1 382-6706
mobile phone number +36 20 210-0880
own web page
Academic title
scientific degree, title Ph.D.
year degree was obtained 2006
discipline to which degree belongs clinical medicine
institution granting the degree SOTE
Employment
2003 - MTA TTK
other (not specified) (tudományos főmunkatárs)
Thesis topic supervisor
number of doctoral students supervised until now 2.5
number of students who fulfilled course requirements 0
students who obtained their degrees:
present PhD students:
Álmos Tilajka (PhD) (2026/08)  DSB-ELTE
Loretta László (PhD) (2024/08)  DSB-ELTE
Tamás Takács (PhD) (2024/08)  DSB-ELTE
  Thesis topic proposals
Research
research area Molecular background of cellular regulatory mechanisms: Our aim is to discover new functions of signaling molecules as adapter proteins, using cellular model systems and gene knockout mouse strains.
research field in which current research is conducted biology
theoretical medicine
Publications
2022

László Loretta, Maczelka Hédi, Takács Tamás, Kurilla Anita, Tilajka Álmos, Buday László, Vas Virag, Apáti Ágota: A Novel Cell-Based Model for a Rare Disease: The Tks4-KO Human Embryonic Stem Cell Line as a Frank-Ter Haar Syndrome Model System, INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 23: (15) 8803
type of document: Journal paper/Article
language: English
URL 
2021

László Loretta, Kurilla Anita, Takács Tamás, Kudlik Gyöngyi, Koprivanacz Kitti, Buday László, Vas Virag: Recent updates on the significance of KRAS mutations in colorectal cancer biology, CELLS 10: (3) 667
type of document: Journal paper/Review paper
number of independent citations: 16
language: English
URL 
2020

Buday László, Vas Virág: Novel regulation of Ras proteins by direct tyrosine phosphorylation and dephosphorylation, CANCER AND METASTASIS REVIEWS 39: (4) pp. 1067-1073.
type of document: Journal paper/Review paper
number of independent citations: 16
language: English
URL 
2019

Vas Virag, Kovács Tamás, Körmendi Szandra, Bródy Andrea, Kudlik Gyöngyi, Szeder Bálint, Mező Diána, Kállai Dóra, Koprivanacz Kitti, Merő Balázs L, Dülk Metta, Tóvári József, Vajdovich Péter, Şenel Ş Neslihan, Özcan Ilknur, Helyes Zsuzsanna, Dobó-Nagy Csaba, Buday László: Significance of the Tks4 scaffold protein in bone tissue homeostasis, SCIENTIFIC REPORTS 9: (1) 5781
type of document: Journal paper/Article
number of independent citations: 4
language: English
URL 
2019

Vas Virag, Háhner Tamás, Kudlik Gyöngyi, Ernszt Dávid, Kvell Krisztián, Kuti Dániel, Kovács Krisztina J, Tóvári József, Trexler Mária, Merő Balázs L, Szeder Bálint, Koprivanacz Kitti, Buday László: Analysis of Tks4 Knockout Mice Suggests a Role for Tks4 in Adipose Tissue Homeostasis in the Context of Beigeing, CELLS 8: (8) 831
type of document: Journal paper/Article
number of independent citations: 1
language: English
URL 
2015

Zirafi O, Kim K-A, Ständker L, Mohr KB, Sauter D, Heigele A, Kluge SF, Wiercinska E, Chudziak D, Richter R, Moepps B, Gierschik P, Vas V, Geiger H, Lamla M, Weil T, Burster T, Zgraja A, Daubeuf F, Frossard N, Hachet-Haas M, Heunisch F, Reichetzeder C, Galzi J-L, Pérez-Castells J, Canales-Mayordomo A, Jiménez-Barbero J, Giménez-Gallego G, Schneider M, Shorter J, Telenti A, Hocher B, Forssmann W-G, Bonig H, Kirchhoff F, Münch J: Discovery and Characterization of an Endogenous CXCR4 Antagonist, CELL REPORTS 11: (5) pp. 737-747.
type of document:
number of independent citations: 37
language: English
URL 
2013

Yolamanova M, Meier C, Shaytan AK, Vas V, Bertoncini CW, Arnold F, Zirafi O, Usmani SM, Müller JA, Sauter D, Goffinet C, Palesch D, Walther P, Roan NR, Geiger H, Lunov O, Simmet T, Bohne J, Schrezenmeier H, Schwarz K, Ständker L, Forssmann W-G, Salvatella X, Khalatur PG, Khokhlov AR, Knowles TPJ, Weil T, Kirchhoff F, Münch J: Peptide nanofibrils boost retroviral gene transfer and provide a rapid means for concentrating viruses, NATURE NANOTECHNOLOGY 8: (2) pp. 130-136.
type of document: Journal paper/Article
number of independent citations: 79
language: English
URL 
2013

Florian MC, Nattamai KJ, Dörr K, Marka G, Überle B, Vas V, Eckl C, Andrä I, Schiemann M, Oostendorp RAJ, Scharffetter-Kochanek K, Kestler HA, Zheng Y, Geiger H: A canonical to non-canonical Wnt signalling switch in haematopoietic stem-cell ageing, NATURE 503: (7476) pp. 392-396.
type of document: Journal paper/Article
number of independent citations: 203
language: English
URL 
2012

Vas V, Wandhoff C, Dörr K, Niebel A, Geiger H: Contribution of an aged microenvironment to aging-associated myeloproliferative disease, PLOS ONE 7: (2) e31523
type of document: Journal paper/Article
number of independent citations: 40
language: English
URL 
2008

Urban VS, Kiss J, Kovacs J, Gocza E, Vas V, Monostori E, Uher F: Mesenchymal stem cells cooperate with bone marrow cells in therapy of diabetes, STEM CELLS 26: (1) pp. 244-253.
type of document: Journal paper/Article
number of independent citations: 290
language: English
URL 
Number of independent citations to these publications:686 
Scientometric data
list of publications and citations
number of scientific publications that meet accreditation criteria:
47
number of scientific publications:
44
monographs and professional books:
0
monographs/books in which chapters/sections were contributed:
0 
scientific publications published abroad that meet the accreditation criteria:
33
publications not in Hungarian, published in Hungary, meeting the accreditation criteria:
2
number of independent citations to scientific publications and creative works:
1082


2024. IV. 17.
ODT ülés
Az ODT következő ülésére 2024. június 14-én, pénteken 10.00 órakor kerül sor a Semmelweis Egyetem Szenátusi termében (Bp. Üllői út 26. I. emelet).

 
All rights reserved © 2007, Hungarian Doctoral Council. Doctoral Council registration number at commissioner for data protection: 02003/0001. Program version: 2.2358 ( 2017. X. 31. )