Tamás Földi - Data sheet of PhD student


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Data sheet of PhD student

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Personal data

name	Tamás Földi
name of institution	University of Szeged
doctoral school	SzTE Theoretical Medicine Doctoral School

Supervision
thesis supervisor	Antal Berényi
one supervisor or with co-supervisor	individual
degree	PhD
starting of doctoral studies	2020/09
foreseen date of final certificate (year,month)	2024/08

Publications

2021

Földi Tamás, Lőrincz Magor L., Berényi Antal: Temporally Targeted Interactions With Pathologic Oscillations as Therapeutical Targets in Epilepsy and Beyond, FRONTIERS IN NEURAL CIRCUITS 15: 784085
type of document: Journal paper/Review paper
language: English
URL

2021

Takeuchi Yuichi, Harangozó Márk, Pedraza Lizeth, Földi Tamás, Kozák Gábor, Li Qun, Berényi Antal: Closed-loop stimulation of the medial septum terminates epileptic seizures, BRAIN 144: (3) pp. 885-908.
type of document: Journal paper/Article
number of independent citations: 4
language: English
URL

2020

Kozák Gábor, Földi Tamás, Berényi Antal: Spike-and-Wave Discharges Are Not Pathological Sleep Spindles, Network-Level Aspects of Age-Dependent Absence Seizure Development in Rats., ENEURO 7: (1) 0253-19.2019
type of document: Journal paper/Article
number of independent citations: 3
language: English
URL

2018

Kozak G, Foldi T, Berenyi A: Chronic Transcranial Electrical Stimulation and Intracortical Recording in Rats, JOVE-JOURNAL OF VISUALIZED EXPERIMENTS 2018: (135) e56669
type of document: Journal paper/Article
number of independent citations: 2
language: English
URL

Number of independent citations to these publications:

Scientometric data

list of publications and citations

number of scientific publications that meet accreditation criteria:

number of scientific publications:

monographs and professional books:

monographs/books in which chapters/sections were contributed:

number of independent citations to scientific publications and creative works:

Research
thesis topic announcement	Many neuropsychiatric disorders are coupled with altered, pathologic oscillations at the neuronal network levels, which prevents proper information processing. While the